Endocrine Society Features Two ME/CFIDS Papers
At the 84th Annual Meeting of The Endocrine Society, held on June 19-22 in San Francisco, two papers on Chronic Fatigue Syndrome (CFS) were presented showing more abnormalities of the "enigmatic disorder, often acquired." One, entitled "Association of Corticosteroid-Binding Globulin Gene Polymorphisms with Chronic Fatigue Syndrome" (Torpy, et al) was work that was done at the Medical University of Queensland, Brisbane (Australia). Studies with twins showed a possible genetic link in monozygotic vs. dizygotic twins and an increased risk of the illness in blood relatives of patients with the illness. Unlike the twin studies funded by the US Government in the United States, the Australian studies have not concentrated their efforts on searching for some thread of psychobabble but have zeroed in on the biology of the cruel illness. The study found that a corticosteroid binding globulin (CBG) increased the risk of acquiring CFS in blood relatives. The CBG is a 383 amino acid corticol carrier protein and it participates in the stress response. Since it fell following an acutely stressful period along with hypercoritsolism, which increased the free cortisol, it was hypothesized the this CBG gene may predispose a person to developing the illness either through an HPA (hypothalamic-pituitary-adrenal) axis means or through an in inherited means. The second paper presented on the subject was entitled "Altered Interrelations among Plasma Stress Hormones during Treadmill Exercise in Females with Chronic Fatigue Syndrome." The authors (Habib et al) were from the Clinical Nuroendocrinology Branch of the NIMH in Bethesda, MD. This project concentrated on the cortisol as well and found "significant abnormalities in the interaction between the sympathetic-adrenal limb and the HPA limb of the stress system in CFS". This suggests that the abnormal catecuolamine metabolism could very well contribute to the deficient stress response in ME/CFIDS patients. |
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